Cardiology Quick Notes - Copyright Dr D O'Kane 2007

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Defect
Causes
Clinical Symptoms
Clinical Signs
Investigations
Management
Aortic Stenosis
normal orifice 3-4 cm2 - impaired LV emptying leading to a pressure overloaded left ventricle
Congenital Bicuspid valve
Rheumatic heart disease
Senile Degeneration
William's syndrome (high calcium)
Chest pain - angina
Dizziness
Syncope
Breathlessness
Endocarditis
Arrhythmias
Sudden death
Slow rising plateau pulse (pulsus tardus et parvus)
Narrow pulse pressure
Harsh Ejection systolic thrill and murmur
Ejection click
Sustained forceful apex
Soft A2
audible S4 (SR)
ECG - SR Left atrial enlargement, LVH + strain, LBBB, AV block
Echo - LVH, gradient, thickened cusps, reduced mobility. Peak gradient > 50 mmHg is significant and suggests moderate stenosis
CXR - normal size + post stenotic dilatation
Cardiac catheter done in those over 40 to look for Coronary artery disease
Avoid exercise if heart failure
Endocarditis prophylaxis
Avoid vasodilators in significant disease
Valve replacement in all with  gradient > 75 mmHg or symptoms and gradient > 50 mmHg
Aortic balloon valvuloplasty - short term solution in those not fit for valve replacement
Replace before decline in LV function
Aortic Incompetence
Blood refluxes back into the left ventricle in diastole creating a volume overloaded left ventricle

(Look for secondary causes and always exclude endocarditis)
Rheumatic heart disease
Infective endocarditis
Connective tissue disease
Degeneration
Bicuspid valve
Endocarditis
Root dilation - hypertension, ankylosing spondylitis, SLE, RA, Aortic dissection

Men > Women.
Breathlessness
Palpitations
Endocarditis
Sudden death
Large volume collapsing pulse with wide pulse pressure - water hammer
High pitched early diastolic murmur best heard at LSE in expiration
Possible added systolic aortic flow murmur + S3
Austin flint murmur - low pitched diastolic AMVL vibrations
Corrigan's sign - visible carotid pulsation
deMussets - head nodding
Quincke's - nailbed pulsations
Traube's - pistol shot femorals
Duroziez's - femoral diastolic murmur
Cardiomegaly - aortic root dilation, LV volume overloaded. Exclude endocarditis. Assess LV
Echo - LV enlargement
Cardiac catheter if valve replacement considered
Vasodilator therapy - ACE, Nifedipine
Diuretics, Digoxin if AF
Aortic Valve replacement if end systolic diameter > 50 mm or signs of LV dysfunction
Mitral Stenosis
normal valve is 4-6 cm2. Narrowing  obstructs and impairs LV inflow and raises Left atrial pressure
Rheumatic heart disease - commoner in women
Congenital
Lutembacher's syndrome
(Left atrial myxoma can mimic Mitral stenosis)
Exertional Dyspnoea
PND
Fatigue
Palpitations
Haemoptysis
Thromboembolism
Hoarseness (Ortner's syndrome)
Dysphagia (pressure of LA)
Frank pulmonary oedema
AF Irregular Irregular pulse
Low volume pulse
Mitral facies
Loud S1 and tapping apex beat
Opening snap
Middiastolic low pitched rumbling murmur increased with exercise
Presystolic accentuation(SR)
Loud P2
Closer the A2-OS the more severe the disease
Pulmonary hypertension - RV lift, Loud P2, TR
ECG - AF and P mitrale, RVH
CXR - normal sized LV and straight left heart border due to increased Left atrial size. MV calcification. Pulmonary oedema
Echo - fish mouth appearance
Severe MS - Reduced MV area to 1 cm2
Catheterisation - raised Left atrial pressure with pulmonary hypertension

Diuretics.
Fast AF - Digoxin, Beta blockers, Verapamil to slow rate and prolong diastolic filling
Warfarin for AF
Mitral commissurotomy
Valvuloplasty
Mitral Valve replacement
Oral penicillin if past rheumatic fever to prevent recurrent rheumatic fever if age < 30

Mitral Incompetence
reflux of blood back into the left atrium during systole due to impaired valve function due to dilation of valve ring or abnormal valve or papillary muscle function
Rheumatic heart disease
Functional (LV dilates)
Endocarditis
Mitral valve prolapse
Myocardial infarction - inferior wall
Myxomatous degeneration
Connective tissue disease
Trauma
Cardiomyopathy (HCM)
Dyspnoea
Palpitations
Fatigue
Endocarditis

(Acute MR leads to a more acute and severe deterioration than chronic MR where there is time for the LA to dilate with time and dampen pressure and volume changes)
Atrial fibrillation
Displaced hyperdynamic apex
PSM at apex often radiates to axilla
Soft S1, Loud P2
Echocardiogram
Cardiac catheterisation
Diuretics
Warfarin for AF
Digoxin for AF
Vasodilator therapy
Mitral valve repair and reconstruction is preferable
Mitral valve replacement if end systolic dimension > 50 mmHg or LVEF < 50%
Endocarditis prophylaxis

Tricuspid Stenosis
Impaired filling of RV due to a narrowed tricuspid valve
Rheumatic disease
Carcinoid
Congenital disease
Right atrial myxoma mimics TS
Usually coexists with mitral valve disease.
Large 'a' wave in JVP
Slow y descent in JVP
Loud S1
Low pitched MDM + PSA, Pulsatile,  Hepatomegaly
Jaundice, Ascites, Pedal oedema
CXR - RAE
Echo - RAE and stenosed valve
Valve repair or replacement in severe cases
Tricuspid Incompetence
blood refluxes from RV back into RA in systole
Functional and Acquired due to pulmonary hypertension (MS, LVF, COAD)
Endocarditis due to IV drug user
Carcinoid
RV infarction and RV dysfunction
Ebstein's anomaly
Right heart failure
Large CV waves
Pulsatile liver
Raised JVP, RV lift,
loud P2,
PSM at LSE increases with inspiration
Oedema
Large V waves in JVP
Pulsatile liver
PSM at LSE louder on inspiration
ECG - AF, P pulmonale, RBBB
Echo - defines valve and helps to determine aetiology, PA pressure indirectly measured. 
Endocarditis prophylaxis
Diuretics
Tricupid valve repair may be undertaken along with mitral valve surgery
Treat any endocarditis
Pulmonary Stenosis
Congenital
Fallot's
Rheumatic
Turner's syndrome

Possibly Dysmorphic
Dyspnoea
Fatigue
Right heart failure
Oedema
Prominent a wave
Ejection systolic murmur 2nd LICS
Increases on inspiration
Ejection click
Soft P2
?Fallot's
P pulmonale, RAD, RVH, RBBB
CXR - post stenotic dilatation of PA, large RA and RV. Pulmonary oligaemia.
Pulmonary valvuloplasty or Valvotomy
Pulmonary Incompetence


Early diastolic murmur
Maximal 2/3rd LICS



Mitral Valve prolapse
Click murmur syndrome - Common cardiac anomaly with many associations - look for those with evidence of MR
Congenital - 2% of men and 5% of women Autosomal dominant trait
Connective tissue disease - Marfan's syndrome
Multiple associations
Nonspecific - causality difficult to prove.
Dyspnoea, chest pain and palpitations all reported.
Mid systolic click and mid to late systolic murmur which increases on standing and with valsalva which both reduce preload
ECG - non specific changes
Echocardiogram
Endocarditis prophylaxis for those with associated MR
Beta blockers
Warfarin if AF develops
Hypertrophic Cardiomyopathy - an autosomal dominant inherited disease with left ventricular hypertrophy and diastolic dysfunction. There is a rare but significant risk of arrhythmias and sudden death in some. Only 20% have LVOT obstruction. Abnormal sarcomeric contractile proteins.
Usually a point mutation in the gene encoding of sarcomeric contractile proteins (actin, Beta myosin, tropomyosin etc)
Dyspnoea - diastolic dysfunction (rise in LVEDP)
Syncope or pre syncope or even sudden death
Palpitations
Sudden cardiac death in a relative
  • Jerky pulse, AF
  • Cardiac failure
  • JVP - prominent 'a' wave due to forceful atrial contraction against a stiff right ventricle + S4
  • Double or even triple apex beat
  • Pansystolic murmur due to Mitral regurgitation
  • Late systolic murmur due to LV outflow track obstruction
  • Increase the murmur by decreasing LV size and volume - Catecholamine excess, Exercise, Inotropes, Digoxin
  • Decrease the murmur - Valsalva, Sudden standing, GTN, Tachycardia - Squatting, handgrip, leg raising to increase venous return, expansion of blood volume
  • HCM may be totally asymptomatic and picked up by screening either with an abnormal ECG - deep T wave inversion, ST and Q wave "pseudoinfarction" appearance, abnormal Echo - Asymmetrical septal hypertrophy, systolic anterior movement of AMVL
  • Implantable defibillator for those at high risk of sudden cardiac death
  • Beta Blockers or Verapamil, disopyramide
  • Surgical myomectomy
  • Non surgical ablation eg injecting alcohol
  • Ventricular septal defect
    abnormal communication between the right and left ventricles. Can be muscular or membranous or subvalvular. Small defects can give loud murmurs.
    Congenital 2/1000
    Acquired (post MI)
    Symptoms depend on size of shunt in terms of blood flow which is a function of VSD size and pressures across the septum.
    Paradoxical embolism
    Cardiac failure
    Eisenmenger's syndrome
    .
    Loud PSM at LSE
    Loud does not mean big VSD
    Wide split S2
    S3
    Middiastolic flow murmur
    ECG - LVH / RVH
    Cardiomegaly, pulmonary plethora
    Echo 2D and doppler echo diagnostic
    Catheter shows increase PaO2 in RV

    Surgical closure before child starts school for moderate sized VSD
    Close if Shunt > 3:1 P/S flow and asymptomatic
    Atrial septal defect
    abnormal communication between the right and left atria - be aware of embryological development. Ostium Primum more severe - associated with endocardial cushion and mitral valve defects. Ostium Secundum - more benign form and commoner
    Congenital
    Down's syndrome and Holt Oram syndrome associated with primum defects

    None, Fatigue,
    Heart Failure,
    Arrhythmias,
    Paradoxical embolism
    Failure to thrive in childhood with Primum defect which is  more severe
    Eisenmenger's syndrome
    Fixed split S2
    Diastolic tricuspid flow murmur
    Systolic pulmonary flow murmur

    Ostium Primum - LAD + RBBB
    Ostium secundum - RAD + RBBB
    Atrial fibrillation
    CXR - large RA, large RV, large PA, Pulmonary plethora
    Catheter shows increase PaO2 in RA
    Warfarin and Digoxin for AF
    Diuretics for heart failure
    Treat surgically if significant shunt before Eisenmenger's syndrome
    usually if pulm/systemic flow is > 1.5:1
    Patent Ductus Arteriosus
    abnormal persisting communication between aorta and pulmonary artery. LV then supplies systemic and pulmonary circulation and volume overloaded LV depends on anatomy of the ductus and pulmonary vascular resistance. Eisenmenger's can develop
    Congenital
    Rubella
    Associated with VSD and CoA
    No symptoms if small
    Endoarteritis of ductus
    High output cardiac failure
    Eisenmenger's syndrome - reversal of shunt - Clubbing and cyanosis of toes and not of fingers
    Continuous murmur best heard in second left intercostal space
    S3
    Mitral flow murmur
    Wide pulse pressure
    ECG - LVH
    CXR - cardiomegaly and pulmonary plethora
    Color-flow doppler
    Catheterisation shows higher SaO2 in PA than RV
    Small PDA inconsequential and not operated if older middle aged asymptomatic
    Surgical division of the defect

    Coarctation of the Aorta (CoA)
    Narrowing of the aorta usually ust distal to the left subclavian artery giving hypertension proximally and under perfusion beyond the stenosis

    Congenital
    Commoner in boys
    Hypertension
    Headaches
    Nosebleeds
    Cold feet

    Systolic murmur maximal over apex left lung posteriorly
    Continuous murmur from collatorals
    LV Heave
    Weak femoral pulse
    Radio-femoral delay
    Upper body hypertension
    ECG - LVH
    CXR - Normal heart or enlarged LV
    Dilated ascending aorta
    Reversed 3 signs - Aortic indentation
    Rib notching from collatorals 2nd to 6 th ribs posteriorly
    Use Echocardiography or MRI
    Measure gradient using cardiac catheterisation
    Surgical correction
    Fallot's Tetralogy - components are
  • Ventricular septal defect
  • Right ventricular outflow obstruction
  • Overriding aorta
  • Right ventricular hypertrophy

  • Congenital

    Heart Failure
    Cyanosis
    Tet Spells
    Eisenmenger's syndrome


    Cyanosis (not initially at birth - may develop)
    Clubbing
    RV Heave
    Thrill at LSE
    Single A2
    Short pulmonary ejection systolic murmur
    ECG - RAD RVH
    Boot shaped heart (absent pulmonary artery gives concavity)
    RV enlarged
    Surgical correction
    Eisenmenger's syndrome
    Right to left shunt due to the development of pulmonary hypertension
    Ventricular septal defect (single S2)
    Patent ductus arteriosus (differential cyanosis)
    Atrial Septal defect (Fixed split S2)
    Cyanosis
    Finger Clubbing
    Polycythaemia
    Dominant 'a' wave in JVP
    Prominent 'cv' wave if TR
    Loud P2
    S4
    Pulmonary ejection click
    Signs of TR and PR





    Dyanmic testing of murmurs

    Condition
    Valsalva
    Standing
    Isotonic exercise
    Squatting
    Raised legs
    Isometric(handgrip)
    Hypertrophic Cardiomyopathy
    Louder
    Louder
    Louder
    Softer
    Softer
    Softer

    Mitral valve prolapse
    Longer murmur

    Louder

    Shorter murmur

    Shorter murmur